Genetic inactivation of p62 leads to accumulation of hyperphosphorylated tau and neurodegeneration.

Retrograde axonal transport and motor neuron disease.

Interaction of amyotrophic lateral sclerosis (ALS)-related mutant copper-zinc superoxide dismutase with the dynein-dynactin complex contributes to inclusion formation.

Interaction of amyotrophic lateral sclerosis (ALS)-related mutant copper-zinc superoxide dismutase with the dynein-dynactin complex contributes to inclusion formation.

[Anonymous].  2008.  Interaction of amyotrophic lateral sclerosis (ALS)-related mutant copper-zinc superoxide dismutase with the dynein-dynactin complex contributes to inclusion formation.. The Journal of biological chemistry. 283(33):22795-805.

Enzymatically inactive adenylate kinase 4 interacts with mitochondrial ADP/ATP translocase.

RABA (reductive alkylation by acetone): a novel stable isotope labeling approach for quantitative proteomics.

Proteomic characterization of lipid raft proteins in amyotrophic lateral sclerosis mouse spinal cord.

Mitochondrial dysfunction in amyotrophic lateral sclerosis.

Prohibitin regulates TGF-beta induced apoptosis as a downstream effector of Smad-dependent and -independent signaling.

Sequestosome 1/p62 links familial ALS mutant SOD1 to LC3 via an ubiquitin-independent mechanism.

Whole genome association study in a homogenous population in Shandong peninsula of China reveals JARID2 as a susceptibility gene for schizophrenia.

Whole genome association study in a homogenous population in Shandong peninsula of China reveals JARID2 as a susceptibility gene for schizophrenia.

[Anonymous].  2009.  Whole genome association study in a homogenous population in Shandong peninsula of China reveals JARID2 as a susceptibility gene for schizophrenia.. Journal of biomedicine & biotechnology. 2009:536918.